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    • Original Article
      Open Access

      Ninjurin1 Deletion in NG2-Positive Pericytes Prevents Microvessel Maturation and Delays Wound Healing

      JID Innovations
      Vol. 2Issue 6100141Published online: July 6, 2022
      • Risa Matsuo
      • Mari Kishibe
      • Kiwamu Horiuchi
      • Kohei Kano
      • Takamitsu Tatsukawa
      • Taiki Hayasaka
      • and others
      Cited in Scopus: 0
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        The formation of mature vasculature through angiogenesis is essential for adequate wound healing, such that blood-borne cells, nutrients, and oxygen can be delivered to the remodeling skin area. Neovessel maturation is highly dependent on the coordinated functions of vascular endothelial cells and perivascular cells, namely pericytes (PCs). However, the underlying mechanism for vascular maturation has not been completely elucidated, and its role in wound healing remains unclear. In this study, we investigated the role of Ninjurin-1 (Ninj1), a new molecule mediating vascular maturation, in wound healing using an inducible PC-specific Ninj1 deletion mouse model.
        Ninjurin1 Deletion in NG2-Positive Pericytes Prevents Microvessel Maturation and Delays Wound Healing
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